45 th dust off air ambulance 1970 -71 crew chief uh1h yes sir there was agent orange being sprayed at long Binh stuff stuck on your arms after the planes come flying over, see them go down highway 1 and all down to Xuan Loc. Glad I made it back to much blood. My head is still there most of the time. I feel the pain
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on long binh 1971 with the 632nd heavy equipment maint.co.worked in the sweat shop you know what i mean.when we stood down i was transfered to the 4th.tc 47th. trans and i was running jp 4 to firebase melanie.18 years old and pulling tankers of that stuff alone.was never afraid full of piss and vinegar.left to go back to the world to fort devens ma.got early out to go in the guards and stayed in for a few years.now almost 68 and 100% agent orange disabled for about five years.severe diabetes and heart disease.did not know why i was sick all the time.had to raise a family so i never went to the doctors.found out about the law passed in 1991 about boots on the ground and i filed all my papers for disability.took them 5 years to work with me,they lost my paperwork took the d.a.v.and the purple heart foundation working for me .they found my files
Trying to find out more about a Colonel Castle, Provost Marshal, Long Binh Post, Lost thumb/KIA/, on LBP perimeter; one tough SOB who last radio contact was; S3 & Driver Dead, blew off my thumb.will probably pass out soon from loss of blood>Did he survive or not? Being Bird Col. could have send subordinates to check perimeter, but went himself: very impressive.
I was at Long Binh 26 Aug 71 to 23 Apr 72. I was the Intelligence NCOIC in the Intel Plans and Opns Branch, DCOPS, HQ USARV. My bosses were COL Farrell and MAJ Tom Rose. Also pulled additional duty as the Intel NCO at the USARV TOC (night shift). I did not spend a full year there but got credit for it. I was caught in the drawdown of US Forces. But not to worry, I went on to complete a 30 year career in Intelligence and the Army saw to it that I got other interesting assignments.
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Second, the compressed air itself is also a serious hazard. On rare occasions, some of the compressed air can enter the blood stream through a break in the skin or a body opening. An air bubble in the blood stream is known medically as an embolism, a dangerous medical condition in which a blood vessel is blocked, in this case, by an air bubble. An embolism of an artery can cause coma, paralysis or death depending upon its size, duration, and location. While air embolisms are usually associated with incorrect diving procedures, they are possible with compressed air due to high pressures. While this seems improbable, even a small quantity of air or other gas in the blood can quickly be fatal.
Bilateral sensorineural hearing loss was first reported in two patients with ADA-deficiency who had been successfully treated with HSCT. Structural and infectious causes were excluded and both patients did not receive any conditioning prior to HSCT, precluding that as a potential cause and implicating the underlying metabolic defect [16]. A high prevalence of bilateral sensorineural hearing loss (58%) was reported in a cohort of 12 patients with ADA-deficiency who had been treated with HSCT [17]. In this study, no relationship was found between deafness and dATP levels.
Transplantation using a MSD or MFD without conditioning early after diagnosis is associated with a good outcome in terms of survival and CD3+ recovery in the first year post HSCT [40], but little is known about the long term outcome and quality of immune reconstitution of patients with ADA-deficient SCID. Extended application of newborn screening for SCID may contribute to improving outcomes moreover in the future by allowing identification of infants with SCID (of all genetic causes) before the development of infections and other complications using the detection of T-cell receptor excision circles (TRECs). TRECs are pieces of DNA excised during development of the T-cell receptor, and are an accurate measure of thymic output. Patients with SCID have a significantly decreased number of TRECs which can be detected using the newborn dried blood spot [55]. Earlier diagnosis and a reduced burden of complications allows for more prompt intervention and improved outcome as studies indicate that siblings diagnosed based on a known family history have a significantly improved survival [56]. Despite the positive outlook in terms of immune reconstitution following HSCT as reported by Hassan et al., the follow-up period in this study was a maximum of 27.6 years (median 6.5 years) [40], and longer follow-up data are required to continue to evaluate the permanency of T-lymphocyte immunity, as initial data suggests that thymopoiesis is limited in unconditioned MFD/MSD HSCTs, which may lead to eventual exhaustion of the T-lymphocyte repertoire [40]. The prospects of GT as a therapeutic option are also promising; particularly with the development of refined vectors and gene editing technology, but further long term follow-up monitoring is needed. Development is also taking place in the improvement of PEG-ADA, with a clinical trial ongoing utilising a recombinant source enzyme as an alternative to the currently used bovine sequences [54]. Although no large prospective studies have been performed examining the outcome of non-immunological defects following definitive treatment, retrospective small reports suggest that neurological, behavioural and audiological defects are not corrected. Further studies are needed to examine if factors such as donor type and conditioning regimen, or type of therapy, influence outcomes in these areas. Further understanding is also needed of the underlying pathogenesis involved in the non-immune systemic manifestations to allow optimal investigation and management as well as to fully decipher between the metabolic effects of ADA-deficiency and effects inflicted by infectious agents.
Next Sipylus, hearing the sound of a quiver in the empty air, let out the reins, just as a shipmaster sensing a storm runs for it when he sees the cloud, and claps on all sail, so that not even the slightest breeze is lost. Still giving full rein, he was overtaken, by the arrow none can avoid, and the shaft stuck quivering in his neck, and the naked tip protruded from his throat. Leaning forward, as he was, he rolled down over the mane and the galloping hooves, and stained the ground with warm blood.
The breeze stirs not a hair, the colour of her cheeks is bloodless, and her eyes are fixed motionless in her sad face: nothing in that likeness is alive. Inwardly her tongue is frozen to the solid roof of her mouth, and her veins cease their power to throb. Her neck cannot bend, nor her arms recall their movement, nor her feet lead her anywhere. Inside, her body is stone. Yet she weeps, and, enclosed in a powerful whirlwind, she is snatched away to her own country: there, set on a mountain top, she wears away, and even now tears flow from the marble.
You might think the Athenian women have taken wing: they have taken wings. One of them, a nightingale, Procne, makes for the woods. The other, a swallow, Philomela, flies to the eaves of the palace, and even now her throat has not lost the stain of that murder, and the soft down bears witness to the blood. Tereus swift in his grief and desire for revenge, is himself changed to a bird, with a feathered crest on its head. An immoderate, elongated, beak juts out, like a long spear. The name of the bird is the hoopoe, and it looks as though it is armed. 2ff7e9595c
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